Title Information
Title
A Quantitative Phosphoproteomic Analysis of the T-Cell Receptor Signaling Pathway Using Novel High-Throughput Approaches
Name: Personal
Name Part
Nguyen, Vinh Hai
Role
Role Term: Text
creator
Origin Information
Copyright Date
2011
Physical Description
Extent
xix, 197 p.
digitalOrigin
born digital
Note
Thesis (Ph.D. -- Brown University (2011)
Name: Personal
Name Part
Salomon, Arthur
Role
Role Term: Text
Director
Name: Personal
Name Part
Brossay, Laurent
Role
Role Term: Text
Reader
Name: Personal
Name Part
Wessel, Gary
Role
Role Term: Text
Reader
Name: Personal
Name Part
Gruppuso, Philip
Role
Role Term: Text
Reader
Name: Personal
Name Part
Sauer, Karsten
Role
Role Term: Text
Reader
Name: Corporate
Name Part
Brown University. BIOMED: Molecular Biology, Cell Biology, and Biochemistry
Role
Role Term: Text
sponsor
Genre (aat)
theses
Abstract
Reversible protein phosphorylation plays a pivotal role in the regulation of cellular signaling pathways. Current approaches in phosphoproteomics focus on analysis of the global phosphoproteome in a single cellular state or of receptor stimulation time course experiments, often with a restricted number of time points. Although these studies have provided some insights into newly discovered phosphorylation sites that may be involved in pathways, they alone do not provide enough information to make precise predictions of the placement of individual phosphorylation events within a signaling pathway. Protein disruption and site-directed mutagenesis are essential to clearly define the precise biological roles of the hundreds of newly discovered phosphorylation sites uncovered in modern proteomics experiments. We have combined genetic analysis with quantitative proteomic methods and recently developed visual analysis tools to dissect the tyrosine phosphoproteome of isogenic Zap-70 tyrosine kinase null and reconstituted Jurkat T-cells. In our approach, label-free quantitation using normalization to co-purified phosphopeptide standards is applied to assemble high density temporal data within a single cell type, either Zap-70 null or reconstituted cells, providing a list of candidate phosphorylation sites that change in abundance after T-cell stimulation. SILAC ratios are then used to compare Zap-70 null and reconstituted cells across a time course of receptor stimulation, providing direct information about the placement of newly observed phosphorylation sites relative to Zap-70. These methods are adaptable to any cell culture signaling system in which isogenic wild type and mutant cells have been or can be derived using any available phosphopeptide enrichment strategy. These studies here have revealed quantitative phosphoproteomic signatures, which may be indicative of the ERK positive feedback pathway and Zap-70-dependent FYN regulation. Follow-up studies were performed and discussed in order to further examine these results. Finally, our group has engaged in our own method development, with the aim of improving our sample preparation procedure for LC/MS/MS analysis. A battery of experiments was performed on pervanadate-stimulated Jurkat T-cells and modifications to our sample preparation procedure were discussed.
Subject
Topic
mass spectometry
Subject
Topic
TCR signaling
Subject
Topic
Jurkat T-cells
Subject
Topic
SILAC
Subject
Topic
label-free
Subject
Topic
hybrid quantitative approach
Subject
Topic
Zap-70
Subject
Topic
FYN regulation
Subject
Topic
method development
Subject
Topic
ERK positive feedback
Record Information
Record Content Source (marcorg)
RPB
Record Creation Date (encoding="iso8601")
20111003
Language
Language Term: Code (ISO639-2B)
eng
Language Term: Text
English
Identifier: DOI
10.7301/Z04Q7S7V
Access Condition: rights statement (href="http://rightsstatements.org/vocab/InC/1.0/")
In Copyright
Access Condition: restriction on access
Collection is open for research.
Type of Resource (primo)
dissertations