Title Information
Title
Biological Consequences of Specific and Promiscuous A-to-I RNA Editing Revealed Through Precise Genetic Engineering.
Name: Personal
Name Part
Savva, Yiannis A
Role
Role Term: Text
creator
Origin Information
Copyright Date
2011
Physical Description
Extent
xviii, 231 p.
digitalOrigin
born digital
Note
Thesis (Ph.D. -- Brown University (2011)
Name: Personal
Name Part
Reenan, Robert
Role
Role Term: Text
Director
Name: Personal
Name Part
McKeown, Michael
Role
Role Term: Text
Reader
Name: Personal
Name Part
Johnson, Mark
Role
Role Term: Text
Reader
Name: Personal
Name Part
Bender, Judith
Role
Role Term: Text
Reader
Name: Personal
Name Part
Carmichael, Gordon
Role
Role Term: Text
Reader
Name: Corporate
Name Part
Brown University. BIOMED: Molecular Biology, Cell Biology, and Biochemistry
Role
Role Term: Text
sponsor
Genre (aat)
theses
Abstract
In eukaryotic cells, informational recoding and RNA interference (RNAi) proceed through structurally diverse double-stranded (ds) RNA molecules. Informational recoding of RNA by the catalytic deamination of adenosine to inosine ensues through the action of ADAR, an RNA editing enzyme. In Drosophila, many mRNAs involved in neuro-transmission serve as site-specific substrates for ADARs via base pairing interactions that generate short imperfect duplexes. However, long double strand RNA molecules exhibiting perfect complementarity can be modified extensively by promiscuous ADAR activity antagonizing RNAi-mediated gene silencing. dADAR also edits its own transcript, but the consequences of this auto-regulation regarding mRNA recoding and RNAi in vivo are not well understood. Using ends-out homologous recombination to bi-directionally interfere with dADAR regulation, I show that auto-editing of dAdar mRNA dramatically remodels the landscape of re-coding events in a site-specific manner. These molecular phenotypes correlate with altered localization of dADAR within the nuclear compartment. In addition, I demonstrate that modifying dAdar auto-regulation affects adaptive complex behaviors. Furthermore, I demonstrate that dADAR can act as an enhancer or suppressor of position effect variegation (PEV) and this action can be bi-directionally modulated via its auto-regulation that alters a single amino acid. Together, these results reveal the in vivo relevance of homeostatic control over post-transcriptional mRNA re-coding events, as well as ADAR�s role in determining long dsRNA fate, a key regulator of heterochromatin formation. The research here highlights a more global function for RNA editing regarding neuronal transcriptomic regulation and emphasizes its pivotal role in organismal fitness.
Record Information
Record Content Source (marcorg)
RPB
Record Creation Date (encoding="iso8601")
20111003
Language
Language Term: Code (ISO639-2B)
eng
Language Term: Text
English
Subject (FAST) (authorityURI="http://id.worldcat.org/fast", valueURI="http://id.worldcat.org/fast/1086255")
Topic
RNA editing
Identifier: DOI
10.7301/Z07S7M1Q
Access Condition: rights statement (href="http://rightsstatements.org/vocab/InC/1.0/")
In Copyright
Access Condition: restriction on access
Collection is open for research.
Type of Resource (primo)
dissertations