- Title Information
- Title
- Analysis of an ACC hypersensitive A. thaliana mutant
- Name:
Personal
- Name Part
- Corcoran, Emma
- Role
- Role Term:
Text
- creator
- Name:
Personal
- Name Part
- DeLong, Alison
- Role
- Role Term:
Text
- advisor
- affiliation
- Brown University. Department of Molecular Biology, Cell Biology, and
Biochemistry
- Name:
Corporate
- Name Part
- Brown University. Undergraduate Teaching and Research Award
- Role
- Role Term:
Text
- research program
- Type of Resource
- still image
- Genre (aat)
- posters
- Origin Information
- Place
- Place Term:
Text
- Providence
- Publisher
- Brown University
- Date Created
(encoding="w3cdtf")
- 2015-08-07
- Physical Description
- Extent
- 1 poster
- digitalOrigin
- reformatted digital
- Abstract
- Ethylene is a gaseous hormone that plays an important role in many cellular
processes in plants, including the promotion of fruit ripening and the inhibition of cell
elongation. We have identified an Arabidopsis thaliana mutant that is hypersensitive to ACC,
an ethylene precursor. This ACC Hypersensitive Hypocotyl 1 (ahh1) mutant has short
hypocotyls on untreated MS1 media and is rescued by AVG treatment. In addition, it shows an
exaggerated response to exogenous ACC compared to the wild-type, Col-0. Our goal is to
characterize this mutant's lesion in the ethylene reception pathway by determining whether
ahh1 follows the "eer" ethylene induction pattern or the canonical hypersensitive pattern.
Using quantitative RT-PCR, we found that ahh1 shows neither "eer" nor canonical induction
behavior. From this information, we conclude that we are characterizing an unmapped ethylene
lesion. Since AgNO3 treatment did not completely inhibit ACC induction in ahh1, the ahh1
mutation likely affects a component after ETR1 in the ethylene reception pathway. Moreover,
ahh1 seedlings show a weak ctr-like phenotype. These data suggest that CTR1 may be affected
in the ahh1 mutant.
- Subject (LCSH)
- Topic
- Ethylene
- Subject (LCSH)
- Topic
- Arabidopsis thaliana
- Identifier:
DOI
- 10.26300/tgeq-kh68