Title Information
Title
Identifying embryonic mechanisms that induce a germ cell fate in sea stars
Name: Personal
Name Part
Fresques, Tara
Role
Role Term: Text
creator
Name: Personal
Name Part
Wessel, Gary
Role
Role Term: Text
Advisor
Name: Personal
Name Part
Creton, Robbert
Role
Role Term: Text
Reader
Name: Personal
Name Part
Freiman, Richard
Role
Role Term: Text
Reader
Name: Personal
Name Part
Dunn, Casey
Role
Role Term: Text
Reader
Name: Personal
Name Part
Wharton, Kristi
Role
Role Term: Text
Reader
Name: Personal
Name Part
Extavour, Cassandra
Role
Role Term: Text
Reader
Name: Corporate
Name Part
Brown University. Department of Molecular Biology, Cell Biology and Biochemistry
Role
Role Term: Text
sponsor
Origin Information
Copyright Date
2017
Physical Description
Extent
xvi, 269 p.
digitalOrigin
born digital
Note: thesis
Thesis (Ph. D.)--Brown University, 2017
Genre (aat)
theses
Abstract
Germ cell specification is required for reproduction in all sexually reproducing animals. During animal development, animals can specify their germ cells through inheritance of maternal molecules or through induction by cell-cell signaling events. Although the ancestral mode of germ cell specification appears to occur by inductive mechanisms it is still not clear how signals selectively induce a germ cell fate. I use the sea star as a model to determine how signaling mechanisms direct germ cell formation. First I use RNA in situ hybridizations to identify when genes associated with germ cell formation are expressed during sea star embryogenesis. I find that the conserved germ cell factors Nanos, Vasa, and Piwi all localize in a germ cell pouch at the larva stage. In addition, Nanos and Vasa mRNA appear to be serially restricted into smaller and smaller embryonic domains during early embryogenesis. One of these restriction events involves Left/Right asymmetry because the germ cell pouch forms on the left side of the embryo. Since Nodal is conserved and required for specification of the Left/Right axis I use a gene perturbation strategy, primarily by injecting a translation blocking morpholino into sea star oocytes, to determine the role that the Nodal signal has on germ cell specification. My results show that Nodal inhibits retention of Nanos and Vasa mRNA in the ventral and right sides of the embryo by inhibiting transcription and by stimulating RNA degradation. In addition, my work suggests that Nodal also inhibits cell morphogenetic events in the ventral and right sides of the embryo required for germ cell pouch morphogenesis. Finally, I use RNA in situ hybridizations to determine when Nanos accumulates in diverse echinoderm species to determine how germ cell specification mechanisms have changed during evolution. My results support the growing consensus that germ cell specification by induction in sea stars represents the ancestral state in echinoderms. Lastly, I speculate which developmental phenomena may be repeatedly required for the evolution of an inherited germ cell specification in all animals.
Subject (fast) (authorityURI="http://id.worldcat.org/fast", valueURI="http://id.worldcat.org/fast/00891773")
Topic
Developmental biology
Subject (fast) (authorityURI="http://id.worldcat.org/fast", valueURI="http://id.worldcat.org/fast/00901339")
Topic
Echinodermata
Subject (fast) (authorityURI="http://id.worldcat.org/fast", valueURI="http://id.worldcat.org/fast/00941280")
Topic
Germ cells
Subject (fast) (authorityURI="http://id.worldcat.org/fast", valueURI="http://id.worldcat.org/fast/01094973")
Topic
Reproduction
Language
Language Term (ISO639-2B)
English
Record Information
Record Content Source (marcorg)
RPB
Record Creation Date (encoding="iso8601")
20170616
Identifier: DOI
10.7301/Z0Z31X3P
Access Condition: rights statement (href="http://rightsstatements.org/vocab/InC/1.0/")
In Copyright
Access Condition: restriction on access
Collection is open for research.
Type of Resource (primo)
dissertations